TY - JOUR
KW - Genetic Diseases, Inborn
KW - Humans
KW - leprosy
KW - Models, Genetic
KW - Phenotype
AU - Feitosa M
AU - Krieger H
AU - Borecki I
AU - Beiguelman B
AU - Rao D C
AB - <p>This study comprised 544 nuclear families with 2,925 individuals tested for the Mitsuda reaction, from the Campinas region in Brazil. Segregation analyses suggest the segregation of a major gene (chi 2(1) = 0.07 - 0.07 = 0, p = 1), by failing to reject the hypothesis of Mendelian transmission and by rejecting the hypothesis of nontransmission of a major gene (chi 2(3) = 0.07 - 0.0 = 0.07, p > 0.99; chi 2(2) = 198.28 - 0.0 = 198.28, p < 0.0001).</p>
BT - Human heredity
C1 - http://www.ncbi.nlm.nih.gov/pubmed/8825460?dopt=Abstract
DA - 1996 Jan-Feb
DO - 10.1159/000154322
IS - 1
J2 - Hum. Hered.
LA - eng
N2 - <p>This study comprised 544 nuclear families with 2,925 individuals tested for the Mitsuda reaction, from the Campinas region in Brazil. Segregation analyses suggest the segregation of a major gene (chi 2(1) = 0.07 - 0.07 = 0, p = 1), by failing to reject the hypothesis of Mendelian transmission and by rejecting the hypothesis of nontransmission of a major gene (chi 2(3) = 0.07 - 0.0 = 0.07, p > 0.99; chi 2(2) = 198.28 - 0.0 = 198.28, p < 0.0001).</p>
PY - 1996
SP - 32
EP - 5
T2 - Human heredity
TI - Genetic epidemiology of the Mitsuda reaction in leprosy.
VL - 46
SN - 0001-5652
ER -