@article{18818,
  keywords = {Genetic Diseases, Inborn, Humans, leprosy, Models, Genetic, Phenotype},
  author = {Feitosa M and Krieger H and Borecki I and Beiguelman B and Rao D C},
  title = {Genetic epidemiology of the Mitsuda reaction in leprosy.},
  abstract = {<p>This study comprised 544 nuclear families with 2,925 individuals tested for the Mitsuda reaction, from the Campinas region in Brazil. Segregation analyses suggest the segregation of a major gene (chi 2(1) = 0.07 - 0.07 = 0, p = 1), by failing to reject the hypothesis of Mendelian transmission and by rejecting the hypothesis of nontransmission of a major gene (chi 2(3) = 0.07 - 0.0 = 0.07, p > 0.99; chi 2(2) = 198.28 - 0.0 = 198.28, p < 0.0001).</p>},
  year = {1996},
  journal = {Human heredity},
  volume = {46},
  pages = {32-5},
  month = {1996 Jan-Feb},
  issn = {0001-5652},
  doi = {10.1159/000154322},
  language = {eng},
}